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DEXEDRINE 10 MG DEXTROAMPHETAMINE

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Dexedrine 10 mg Online

Dexedrine 10 mg or Dextroamphetamine is the dextrorotary stereoisomer of the amphetamine molecule. Also, which can take two different forms.  Slightly polar, weak base and is lipophilic.

Dexedrine Spansule

Generic Name: dextroamphetamine (DEX tro am FET a meen) Brand Name: Dexedrine Spansule, ProCentra and also Zenzedi

Other Names of Dexedrine 10 mg

  • (+)-(S)-amphetamine
  • (+)-amphetamine
  • (+)-α-methylphenethylamine
  • (+)-α-methylphenylethylamine
  • (S)-(+)-amphetamine
  • (S)-(+)-β-phenylisopropylamine
  • (S)-1-phenyl-2-aminopropane
  • (S)-1-phenyl-2-propylamine
  • (S)-amphetamine
  • (S)-α-methylbenzeneethanamine
  • (αS)-α-methylbenzeneethanamine
  • d-amphetamine
  • Dexamfetamina
  • Dexamfetamine
  • Dexamfetaminum
  • Dexamphetamine
  • Dexanfetamina
  • Dextroamphetamine

Knowledge about Dextroamphetamine Capsules

Amphetamines such as dextroamphetamine are noncatecholamine. Also, Sympathomimetic amines with CNS stimulant activity. Therefore, Peripheral actions include elevations of systolic and diastolic blood pressures and weak bronchodilator and respiratory stimulant action. Also, There is neither specific evidence that clearly establishes the mechanism. Whereby, Amphetamines produce mental and behavioral effects in children. Finally, No conclusive evidence regarding how these effects relate to the condition of the central nervous system.

Mechanism of Dexedrine

Furthermore, The exact mechanism of action is not known. Therefore, Dextroamphetamine stimulates the release of norepinephrine from central adrenergic receptors. Also, At higher dosages, causes the release of dopamine from the mesocorticolimbic system and the nigrostriatal dopamine systems by reversal of the monoamine transporters. Furthermore, Dextroamphetamine may also act as a direct agonist on central 5-HT receptors and may inhibit monoamine oxidase (MAO). Moreover, In the periphery, amphetamines are believed to cause the release of noradrenaline by acting on the adrenergic nerve terminals and alpha- and beta-receptors. In conclusion, Modulation of serotonergic pathways may contribute to the calming effect.

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